Sammanfattning
Conclusions: Our results suggest that SKIP interacts with AR in the nucleus and enhances AR-dependent transactivation and N/C-interaction supporting a role for SKIP as an AR co-factor.
Originalspråk | Odefinierat/okänt |
---|---|
Sidor (från-till) | 1–9 |
Antal sidor | 9 |
Tidskrift | BMC Biochemistry |
Volym | 14 |
Nummer | 10 |
DOI | |
Status | Publicerad - 2013 |
MoE-publikationstyp | A1 Tidskriftsartikel-refererad |