Sammanfattning
Conclusions: We propose a self-amplifying loop of autocrine S1P with capacity to enhance cell survival. The mechanism provides increased understanding of the multifaceted roles of S1P in regulating cell fate during normal development and carcinogenesis.
Originalspråk | Odefinierat/okänt |
---|---|
Sidor (från-till) | 1–11 |
Antal sidor | 11 |
Tidskrift | BMC Cell Biology |
Volym | 11 |
DOI | |
Status | Publicerad - 2010 |
MoE-publikationstyp | A1 Tidskriftsartikel-refererad |