Abstract
Conclusions: Our results suggest that SKIP interacts with AR in the nucleus and enhances AR-dependent transactivation and N/C-interaction supporting a role for SKIP as an AR co-factor.
Original language | Undefined/Unknown |
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Pages (from-to) | 1–9 |
Number of pages | 9 |
Journal | BMC Biochemistry |
Volume | 14 |
Issue number | 10 |
DOIs | |
Publication status | Published - 2013 |
MoE publication type | A1 Journal article-refereed |